Research

Research projects

Our laboratory explores the physiological role of endoplasmic reticulum (ER) homeostasis in the context of metabolic disorders including, but not limited to, obesity, type-1/-2 diabetes and etc. Our goal is to uncover new findings, break new grounds, delineate the etiology and pathogenesis of human diseases, and eventually help develop therapeutic strategies. In the past 16 years, using cellular, immunological and molecular biology approaches, we have made some important discoveries and produced new insights into the pathogenesis of these diseases. 

PHYSIOLOGICAL ROLE OF ER-ASSOCIATED DEGRADATION (ERAD) IN HEALTH AND DISEASE

We were the first to demonstrate SEL1L as an indispensable component of HRD1 ERAD complex in mammals in vivo (PNAS 2014), discover novel links between SEL1L-HRD1 ERAD and a number of disease-associated proteins and pathways including IRE1a, prohormones and lipoprotein lipase (Nat Cell Biol 2015; Mol Biol Cell 2016; J Clin Invest 2017, 2018; Cell Metabolism 2014), report how ERAD regulates mitochondrial function (Science 2020), and demonstrate how ERAD function in many different tissues and cell types (Nat Cell Biol 2013, 2023; Nat Comm 2023; J Clin Invest 2020, 2021, 2023; EMBO 2019; Cell Reports 2016)... 

Our achievements build on our desire to think outside the box, take the risk and develop breakthrough science. We are at the forefront of the ERAD field. We are uniquely positioned because of many SEL1L-HRD1 ERAD deficient cell and mouse models that we have recently generated, because of our innovative spirits, passion and dedication to scientific discovery and because of our diverse research expertise in many different areas. 

We will tackle many intriguing questions in the field, in particularly, (a) how different cell types deal with protein misfolding and manage ER homeostasis under various disease settings in mouse models and humans, (b) how ERAD is linked to other signaling pathways including innate immunity and nutrient metabolism in vivo, and (c) biochemically how ERAD works in the cell as well as the nature of endogenous ERAD substrates in a cell type specific manner. These studies will likely have significant impact as they will fundamentally change our views on the role of ER homeostasis, ERAD and protein folding and degradation in physiology and diseases.

Lab in the News

University of Virginia (2023-present)

 

05/03/23 Medicine in Motion News, UVA  

Ling Qi, PhD, to Chair UVA’s Department of Molecular Physiology and Biological Physics

https://news.med.virginia.edu/featured/ling-qi-phd-to-chair-uvas-department-of-molecular-physiology-and-biological-physics-3/

 

 

University of Michigan (2016-present)


3/30/20  University of Michigan Health Lab   Unlock the secrets of brown fat

https://labblog.uofmhealth.org/lab-report/unlocking-secrets-of-brown-fat

 

5/20/19  University of Michigan Health Lab   Discovery in mice could remove roadblock to more insulin production

https://labblog.uofmhealth.org/lab-report/discovery-could-remove-roadblock-to-more-insulin-production

 

1/19/18  University of Michigan Health Lab   Clues to Obesity’s Roots Found in Brains’ Quality Control Process https://labblog.uofmhealth.org/lab-report/clues-to-obesitys-roots-found-brains-quality-control-process

 

9/18/17  University of Michigan Medicine      Urine output to disease: study sheds light on the importance of hormone quality control

https://www.eurekalert.org/pub_releases/2017-09/mmu-uot091517.php

 

11/16/16 University of Michigan Health Lab   Researching Proinsulin Misfolding to Understand Diabetes

http://labblog.uofmhealth.org/industry-dx/researching-proinsulin-misfolding-to-understand-diabetes


Cornell (2007-2016)

11/12/15    Cornell Chronicle      Mechanism underlying cell stress response discovered  (http://www.news.cornell.edu/stories/2015/11/mechanism-underlying-cell-stress-response-discovered)

8/4/14    Cornell Chronicle      A new player in lipid metabolism discovered (http://www.news.cornell.edu/stories/2014/08/new-player-lipid-metabolism-discovered)

6/18/14    Cornell Chronicle     Chronic intake of Western diet kills mice  (http://www.news.cornell.edu/stories/2014/06/chronic-intake-western-diet-kills-mice)

5/28/14    Cornell Chronicle     20 Cornellians win SUNY Chancellor’s Awards for Excellence (http://www.news.cornell.edu/stories/2014/05/20-cornellians-win-suny-chancellors-awards-excellence)

1/22/14    Cornell Chronicle     Gene prevents buildup of misfolded cell proteins  (http://www.news.cornell.edu/stories/2014/01/gene-prevents-buildup-misfolded-cell-proteins)

 8/21/13       ABC News        There’s no perfect diet, researcher says  (http://abcnews.go.com/Health/stop-diet-madness-researcher/story?id=20023902)

5/13             Diabetes Forecast       Powering Diabetes Research (with a profile of Dr. Ling Qi) (http://www.diabetesforecast.org/2013/may/powering-diabetes-research.html?referrer=https://www.google.com/

12/20/12    Cornell Chronicle    Researchers link protein known for cell mobility with protein folding during stress  (http://www.news.cornell.edu/stories/Dec12/ERStress.html)

5/7/12        Cornell Chronicle    Immune cells found to counter obesity-related diabetes (http://www.news.cornell.edu/stories/May12/QiTCells.html)

3/14/12      Cornell Daily Sun    The Scientist: Prof. Ling Qi Examines Fat Cell Responses to Obesity and Diabetes  (http://cornellsun.com/node/50559)

1/31/12      Cornell Chronicle    Qi wins prestigious American Diabetes Association award (http://www.news.cornell.edu/stories/Jan12/QiDiabetesAward.html)

8/17/11      Cornell Chronicle    Grad student wins Hughes fellowship for doctoral research (http://www.news.cornell.edu/stories/Aug11/ShengyiSunAward.html)

 6/2/09       Cornell Chronicle      Cornell researchers discover key regulator of fat cell development   (http://www.news.cornell.edu/stories/2009/06/researchers-discover-pathway-implications-obesity)

6/5/08       Cornell Chronicle       Cornell researcher strives to break the link between obesity and diabetes   (http://www.news.cornell.edu/stories/June08/obesity.diabetes.mw.html)